Resveratrol ehance wound healing activity of the immune system
Enhance wound healing activity of the immune system
Resveratrol can also enhance the immune system and promote burn healing. Some studies of resveratrol in the recovery of burn mice by inhibiting cellular, humoral and non-specific immune function, the effect of resveratrol provides a controlled drug dependence using the type of immune regulation. Severe burns of varying degrees, a study in mice, resveratrol restore their damaged functions, such as the ability to respond to the signal of antigen, proliferative capacity, interleukin-Ⅱ synthesis and antibody synthesis by lymphocytes. Severely burned animals using controlled resveratrol treatment, the level of neutrophil adhesion rate restored to near its normal level, and survival time.
Antioxidant, anti-free radical
Resveratrol is present in plants of natural antioxidants, mainly through the removal or inhibition of free radicals, inhibit lipid peroxidation, antioxidant-related activity regulation mechanisms play an antioxidant role. Most of polyhydroxy stilbene substances have antioxidant, anti-free radical. When resveratrol in 1.3μg / mL, can inhibit the oxidation of rat red blood cells from H2O2-induced hemolysis and the oxidation of hemolysis of mouse heart, liver, brain, kidney and in vivo lipid peroxidation produce significantly inhibited. Resveratrols antioxidant, free radical induced addition of arachidonic acid metabolism and effects of the pharmacological features attracted widespread interest, as these related to metabolism are closely related to human health in many physical diseases such as atherosclerosis , Alzheimers disease, viral hepatitis, gastric ulcer, inflammation and allergic reactions.
Anti-inflammatory, antibacterial
Resveratrol against Staphylococcus aureus, the card he aureus, Escherichia coli, Pseudomonas aeruginosa was inhibited, and the orphan virus, herpes simplex virus and enterovirus, Coxsackie A, B group has a strong inhibitory . Resveratrol by reducing platelet adhesion, the process of change in the anti-inflammatory to anti-inflammatory activity of platelets.